| 저자 |
Yun Kuy Oh1, 5, Chul Woo Yang2, Yong-Lim Kim3, Shin-Wook Kang4, Cheol Whee Park2, Yon Su Kim5, Eun Young Lee6, Byoung Geun Han7, Sang Ho Lee8, Su-Hyun Kim9, Hajeong Lee5, Chun Soo Lim1, 5 |
| 초록 |
Backgroud: Renal infarction (RI) is an uncommon condition resulting from an acute disruption of renal blood flow. The etiology and outcome of RI is not well established
Methods: We retrospectively surveyed the medical records of 438 patients admitted with RI to eight referral hospitals of Korean from January 1993 to December 2013.
Results: Etiology of RI was divided into the following groups: cardiogenic (n=244), idiopathic (n=132), renal artery injury (n=33), and hypercoagulable (n=29). Four hundred and thirty-seven patients were diagnosed by computed tomography. Twenty-one patients received urokinase infusion, 350 patients received heparin, and 335 patients received warfarin. One hundred and sixty one patients were given anti-platelet agents. Fourteen patients were recurred and 90 patients developed acute kidney injury (AKI). Eighty patients progressed to chronic kidney disease (CKD) and nine patients received long-term dialysis therapy. Mortality was 5.0%. Being male (OR, 2.21; 95% CI, 1.03-4.72), systolic blood pressure ((SBP) OR, 1.01; 95% CI: 1.00-1.03), and AKI (OR, 17.82; 95% CI, 8.50-37.33) were associated with CKD. The idiopathic group (OR, 0.29; 95% CI, 0.12-0.68) was related to a lower risk of CKD. Albumin level (OR, 0.39; 95% CI, 0.16-0.96) and warfarin treatment (OR 0.01; 95% CI, 0.00-0.11) were significantly associated with lower mortality.
Conclusion: Cardiogenic origins were the most important causes of RI. Being male, high SBP, and AKI were associated with CKD. The idiopathic group is associated with lower risk for CKD. Lower mortality after RI was associated with higher albumin level and warfarin treatment. |